New findings in mitochondrial biology thoroughly change the idea scientists had for 20 years on the role and importance of the protein MTERF1. For the first time, Max Planck researcher Mügen Terzioglu and her colleagues in Germany and Sweden investigated in vivo what was up to now only explored in cell culture. Using the mouse as a model organism, she made a surprising discovery: MTERF1 does after all not play the key role in mitochondrial transcription and translation that was hitherto ascribed to it.
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